• Vol. 38 No. 7, 630–635
  • 15 July 2009

Association Between Rectosigmoid Adenomas and Cardiovascular Risk Factors: A Hospital-based, Cross-sectional Study

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ABSTRACT

Introduction: Little was known about the association between colorectal adenomas and cardiovascular risk factors in Taiwan. The aim of this study was to assess the association between rectosigmoid adenomas and related factors. Materials and Methods: This was a hospital-based, cross-sectional study. We analysed subjects receiving self-referred health examinations at 1 medical centre in Taiwan between 2001 and 2004. In total, 4413 subjects were enrolled in this study. There were 2444 men (55.4%) and 1969 women (44.6%). The mean age was 49.3 ±12.3 years (range, 20 to 87). All subjects underwent a 60-cm fl exible sigmoidoscopic examination and laboratory survey. Adjusted odds ratio (OR) and 95% confi dence interval (CI) were expressed using a multivariate logistic regression analysis. Results: In the fi nal model, increasing age (OR, 1.05; 95% CI, 1.03-1.06), hypertriglyceridemia (OR, 1.49; 95% CI, 1.07-2.07), and alcohol consumption (OR, 2.11; 95% CI, 1.47-3.04) were the risk factors for rectosigmoid adenomas in men. Increasing age was the only risk factor for rectosigmoid adenomas in women (OR, 1.03; 95% CI, 1.01-1.06). Conclusion: Age, hypertriglyceridemia and alcohol consumption are associated with rectosigmoid adenomas in men, and only age is signifi cantly associated with rectosigmoid adenomas in women.


Lymphatic system returns proteins, lipids and accompanying water from the interstitium to the venous circulation near the subclavian vein-internal jugular vein junction, bilaterally. Lymphoedema results from impaired lymphatic transport leading to the pathologic accumulation of protein-rich lymphatic fluid in the interstitium, most commonly in the extremities.1 Lymphoedema may be classified as primary or secondary, based on underlying aetiology. Primary lymphoedema, a developmental abnormality of the lymphatic system, not always clinically evident at birth, may become evident later in life when a triggering event or worsening of the condition causes the lymphatic transport capacity to exceed the volume of interstitial fluid formation, causing the patient to be unable to maintain normal lymphatic flow.2,3

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